Transient Global Amnesia is aimed at any clinician with an interest in, or who encounters patients with, acute amnesia: neurologists, general physicians, old age psychiatrists, geriatricians, clinical neuropsychologists, and primary care physicians, as well as other professions allied to medicine with similar interests, such as members of memory assessment teams. Based in part on the author’s experience of TGA cases over 20 years and in part on a review of the published literature, this book will hopefully enlighten clinicians from a broad range of medical backgrounds on the clinical features, investigation, and pathogenesis of TGA. During an episode of transient global amnesia, a person is unable to create new memory, so the memory of recent events disappears. Sporadic reports of TGA occur very rarely in a variety of circumstances such as dobutamine-atropine stress echocardiography, infusion of DMSO-cryopreserved autologous peripheral blood stem cells, breathing of. Hence, any history of drug-related amnesia may help clarify mitigating causes. This confused state isnt caused by a more common neurological condition, such as epilepsy or stroke. More than 50 years after its initial description, transient global amnesia (TGA) remains one of the most enigmatic syndromes in clinical neurology. Excessive alcohol can cause a blackout phenomenon. Each of the chapters is devoted to a practical and structured overview of the particular topic, with use of case studies to illustrate the material. Transient global amnesia is an episode of confusion that comes on suddenly in a person who is otherwise alert. Subsequent chapters then discuss the investigation and predisposing and precipitating factors for TGA before attempting to synthesise these many strands in order to formulate ideas about the pathogenesis of TGA. First, it deals with the history of TGA before moving on to clinical and diagnostic features and differential diagnosis. 1 Orientation in space and time is impaired while consciousness remains undisturbed. Global means that the loss of memory is severe and often. This extensively revised second edition provides a comprehensive overview of transient global amnesia (TGA). Patients show reversible MRI signal abnormalities in the CA-1 sector of the hippocampal cornu ammonis early in the course of TGA. Damasio et al (see record 1984-15362-001) differed from the usual case of transient global amnesia because of. Transient global amnesia (TGA) is characterized by anterograde memory disturbance of sudden onset that lasts for 1 to 24 hours. Transient indicates that what happens is brief and, importantly, that the symptoms resolve completely.
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